prevailing truth an accepted truth in mainstream medicine and in our society
as a whole that cholesterol is bad that saturated fat is bad that saturated fat
increases cholesterol and that's why saturated fat is bad and also that high
levels of cholesterol causes heart disease and then basically we've been
told this so many times that we don't question it anymore that the case is closed this is how it is and we're going
to talk about all of these different factors and when I talk about it in a way that you learn to think about it not
just refer to some study or some article but you'll understand these things and
when you think about them you'll be able to go hey that makes sense or that does
not make sense but how come this became the prevailing truth the mainstream
truth the current Dogma and a lot of it has to do with the fact
that we associate things that these fatty plaques have some cholesterol in
them and therefore we think hey that must come from somewhere it must be from what we're eating it must be the
cholesterol and a lot of that thinking had to do with something called the seven countries study so this was late
50s early 60s where a guy called Ansel Keys who was a nutritionist
was very convincing in his arguments so here is how that study came about he
studied number of countries and as you soon notice there were a lot more than
seven countries and in fact there were 22 countries that he started with and
then you would think this should be called the 22 countries study but he
selected a number of these countries so these were the only countries that ended
up in the study which is five of them and then the other countries were
scrapped because they didn't fit what he was looking for so he was had this idea
that he was trying to prove that diets high in saturated fat and cholesterol in
countries where they had those diets they also had more heart disease so he had to pick the countries that match
what he was trying to prove and that was only five countries out of 22 that
matched his criteria and then he had to go and find two more countries Greece
and Yugoslavia so now he had seven countries where he could kind of see a pattern and that's what he published as
the seven country study and you don't have to be a rocket scientist to see that cherry picking like that is not a
great idea if you want to be objective but even so this has been hailed as a
landmark study and is basically the foundation of why we think the way we do
today and since then there's been hundreds of studies performed on these
topics and they tend to kind of fall 50 50 or in in two opposite camps not
necessarily equally split but there are some studies that prove that show that
high cholesterol troll is strongly correlated with cardiovascular disease
but for every study that says that there is another that says there there's no
correlation or even an inverse correlation that actually high cholesterol High saturated fat diets
result in less cardiovascular disease and that's why he had to scrap most of
those 22 countries because a lot of them actually showed the opposite relationship but why is it so difficult
to figure this out if we have gold standard research double-blind Placebo
control why can't they figure it out and one thing that we have to ask is who
pays for it and these studies are not cheap they are in the millions or tens
of millions of dollars to perform a big comprehensive well designed study so if
you have tens of millions of dollars sitting around you can go and order yourself a study and someone will be
happy to perform it for you but for the most part the people or the companies
with that kind of money tend to be pharmaceutical companies so they have huge budgets because it's in their best
interest to prove that high cholesterol causes heart disease but here's an
astounding fact that most people don't realize but it kind of makes sense if you understand human nature we don't
want to look bad so if someone orders a study and they get the results back and
they're favorable then there's an 85 percent chance that their studies will
get published if they can get it through the peer review process to show that it's well designed and Rel relevant then
85 percent of those will get published but if it is not favorable if the results
coming back do not support what they wanted then there's an 85 percent chance
that it won't get published and these are not totally exact numbers because it's going to vary a little bit but
they're roughly in the ballpark and what this shows us is that what we call
scientific truth that we base this on double-blind placebo-controlled studies and we think
hey what's out there what's published that represents the truth but what's
built into this process is a 45x bias
toward the person who ordered the entity who ordered the study so we can't just
look at what's out there and assume that that's the whole truth and nothing but the truth so I don't have tens of
millions of dollars sitting around either so I have performed some of my own studies on a smaller scale and you
may have seen those videos where I do something I get some blood work then I
eat something and then I do blood work again and then I report what happens so
for example I ate 100 eggs in seven days and the pre and post blood work showed a
few items going up or down but overall I did not get sicker there were no dramatic markers that I had a poor
health after doing that then another time I ate a hundred tablespoons of butter in 10 days and interestingly you
would think that all that saturated fat would end up in my bloodstream as triglycerides but my triglycerides went
down and my insulin resistance improved after doing that I ate a hundred
hamburgers in 10 days or rather a hundred hamburger patties because I
didn't want to eat a bunch of bread and carbohydrate to screw up my metabolism
and interestingly my cholesterol went from 233 to 199 which is one of the
lowest ones I've had in recent decades and I don't really know why I thought it
would stay pretty much the same and another interesting thing was the blood urea nitrogen the bun which went from 16
to 10 even though I ate almost twice as much protein as I usually do and the
residue from protein is this nitrogen that we can measure in blood urea nitrogen and when I mentioned in that
video that I wasn't sure why that Bond went down and I still don't one of my
viewers enlightened me and said of course it went down because you ate the
hamburger without the bond and if you notice I put quotation marks around the
word studies because I do that kind of tongue-in-cheek I don't believe that
these are good examples of research this is actually pretty bad science and I'm
not claiming that it's anything else and the reason is that the total sample population meaning the number of people
studied is exactly one namely myself and it still costs a few hundred bucks to do
that so the reason it's not great science is
that I may not be like you whatever results I get could be different than
yours but I'm not claiming that it's good science or that I represent you but
guess what when they do bigger studies if they have a study with 10 000 people
in them and then they have 55 percent of people respond a certain way that's
going to create a trend it's going to be statistically significant probably even
though 4 500 people out of the 10 000 didn't respond that way and I'm just making
coarse gross examples here to illustrate something but the point is that it
doesn't matter how big the study is it may still not be like you okay so you
still have to figure out you and the way that you do that is in these videos I
explain the mechanisms and the basic physiology so you can start understanding how the Body Works rather
than just looking at one example whether it's mine or somebody else's and then
you can look at your own blood work or you can look at your own results in life how you feel your weight your waistline
Etc and then you figure you out but you
can't do that unless you understand some mechanisms and some basic physiology
let's look at some very simple facts eggs have cholesterol eggs have
saturated fat but the question then is first if we eat a bunch of eggs does
this mean that we raise our blood cholesterol we don't know sometimes we do sometimes
we don't but if it did go up would that be a bad
thing and here's the huge misconception that a lot of people think if it goes up
then that's bad but in itself it doesn't mean anything we have to look at the
bigger picture now let's take a look at the concept of conversion of turning one thing into another because based on the
prevailing truth about eggs cholesterol saturated fat and heart disease as a
culture we believe that dietary cholesterol leads to plaques leads to
atherosclerosis so let's take an example let's make a comparison so here we have
an egg which has obviously cholesterol and then we have this theory that this
leads to plaquing and atherosclerosis and
narrowing of arteries and this is basically how we think based on these
studies the seven country studies that the cholesterol from here ends up as
cholesterol in the plaque it's the same cholesterol more of this leads to more
of that but here is a question now if we assume that maybe this isn't true maybe there's
some conversion going on and let's ask a question that if in another example that
we have a cow and
this cow has a body with some fat in it with some
saturated fat with cholesterol in the fat this cow produces milk from which we
can make butter that has lots of fat lots of saturated fat lots of
cholesterol so if this cow is full of cholesterol where did that come from is
it because that cow ate a bunch of eggs with cholesterol did it eat a bunch of
butter with cholesterol did it eat a bunch of meat does it go around eating
the other cows is that how the cholesterol ends up in the cow no if
this cow eats what it's supposed to then all it eats is grass
green grass pure carbohydrates basically it has a trace amount of protein and and
fat in it but basically this cow eats things eats grass that has no cholesterol and yet it ends up with all
that cholesterol in the body so maybe if we understand that there's a conversion
process maybe that cholesterol in the plaque doesn't come from there that's a
possibility and this conversion is also known as metabolism which means to
transform so it's a form of transformation so if we look at the things that we eat as humans and we are
by the way quite similar to a cow in that we are both mammals we both store
fat and we store it as about half saturated
and about half mono unsaturated that's typical for the body fat or both a cow and a human so the big groups the big
macronutrients that we eat are protein carbohydrates and fat and protein the
purpose is to turn into tissue to building blocks to body parts but the excess will actually turn into
carbohydrates and carbohydrates in turn can become fat but it can't work the
other way around so we can convert things sometimes one way but not necessarily both ways and then all three
of these turn into something called acetyl COA
and don't worry about the name don't try to memorize any of this I'm just trying to show you that we have something
called conversion metabolism transformation we eat these things and along the way they become something else
this is an intermediary metabolite that we use to make energy so if we make
energy from protein carbs or fat it's because we can turn it into acetyl COA
but if we're really really low on carbs this acetyl COA can turn into something
else depending on circumstances depending on what's appropriate for the body what it needs at that time so if
we're low on carbs we can turn this acetyl-coa into ketones and this acetyl-coa is also the raw material for
cholesterol so again I'm just trying to show you that this cholesterol didn't
necessarily come from cholesterol that we can turn anything that we eat can
turn into cholesterol if we need it but here's the thing it's really not a mystery anymore today we know what the
cause of cardiovascular disease is and it is chronic inflammation oxidative
stress and something called insulin resistance I'm not going to go into depth here because I've done videos on
all of this and it's also something called metabolic syndrome or Syndrome X
and this is a label this is a cons steps when we're metabolically unhealthy then
we have certain manifestations and if we have abdominal obesity
hypertension meaning high blood pressure if we have high fasting glucose if your
body can't control blood glucose as in type 2 diabetes if we have high
triglyceride high blood fats also very common in diabetes if we have low HDL the supposedly good
cholesterol which of course there is no good or bad there's just appropriate and
appropriate cholesterol then if you have any three out of these five then they
consider you to have metabolic syndrome and then they know that you'll be
predisposed to all these different problems of heart disease Etc
but the people with metabolic syndrome what
they really have is chronic inflammation oxidative stress and insulin resistance so these three things are the causes
that we need to work on and that we need to control and this is very very well
established there's really no discussion or controversy about this at all what there still is controversy about is
which type of foods and what type of Lifestyle will create these states that
cause cardiovascular disease but the question now in this video is do eggs
cause cardiovascular disease so the question is do eggs cause these three
things and the answer is no they don't there is nothing in there that causes
insulin resistance or oxidative stress or inflammation unless you are allergic
to them then that becomes a bad food for you or if you combine the eggs with things
like sugar margarine and chemicals and this is the problem with a lot of
observational studies like the seven country studies that he picked countries
that had a high intake of saturated fat and cholesterol but what else did they
eat did they eat a bunch of sugar margarine and chemicals how much did they smoke do they ever exercise we
don't know but here's what we do know High total cholesterol in itself does
not cause heart disease and here's a article that I've referenced once before but it's so good I want to bring it back
so basically they looked at the total cholesterol levels they only measured
two things they measure total cholesterol and how many people died and
this was a huge study because they did this in Korea over a decade or so and it
was basically everyone in Korea more or less so 12.8 million adults were part of
this study and it's not like the seven country studies where they don't know
all the variables because here they were only interested in two they didn't care
what else they did they only looked at what happens with cholesterol and how
many people die of all the people who died what were their cholesterol levels so they looked at
12.8 million adults and they looked at all cause mortalities what that means is they didn't really care if the
cholesterol caused heart disease or not all they know because you could maybe
save a few people from heart disease but if they die faster from something else then that's not really a gain is it so
all cause mortality is pretty much what we're interested in and here's the interesting thing that they found that
we're told that high cholesterol is bad low cholesterol is good and they give
people statin drugs I've had people come through my office on statin drugs with the cholesterol under 120 and they keep
giving them the stat drug as if less is always better but here's what we find on
this graph is that low cholesterol is really really bad like really low
cholesterol is really bad and if we compare that to very high cholesterol we
see that a cholesterol of of 110 120 is many many times worse much more likely
to cause death than a cholesterol of 300 but when was the last time you saw a
commercial for a medication to raise cholesterol it's like oh my God your cholesterol is 120 we got to get we got
to get you up into the 250s they don't do that so much do they because we have
this bad idea that less is always better and in fact what we found in this study
was that The Sweet Spot the absolute lowest death rate the optimal
cholesterol would fall somewhere around 2 30. and if we looked at the area where
they had a very slight increase above the lowest rate of death like less than
10 percent increase above that lowest rate we would consider that a pretty good pretty good odds then that gives us
a pretty wide range where we are safe so anywhere from 180 to 270 is a very
slight increase in risk and then we overlap that with the standard range
with the normal recommendations that we get all over the world is that the
cutoff is a 200 anything over 200 is too high and you should take medication to
control it and if you notice here that what the study found to be the optimal
range where you have just a slight increase Falls almost entirely in the red area
where you would be prescribed or recommended medication or lifestyle changes or both to lower your
cholesterol now let's just add one more factor to this because I think it's a great study but remember they only look
at the total cholesterol and the total amount of death all-cause mortality but
what we have to realize is that some of these people with high cholesterol were
healthy and some of them were very unhealthy and the other things that we
just talked about that we know cause heart disease that some of these people
have those factors and some didn't so what if we were to take this population
and I'm speaking hypothetically now and we took all the people with abdominal
obesity and we took them out of here because we're trying to find out how does cholesterol affect death if we now
correct correct for the people who had abdominal obesity which we know is a
risk factor and we also take out the people who have hypertension and high
fasting glucose high triglycerides and low HDL we know these are risk factors
for heart disease let's take these people out so now we isolate more for
what cholesterol in itself actually does and we also correct we take the people
out who have high insulin levels now we have a pure population where only
cholesterol is a causative factor of death and I believe
not that I've ever known that they've done this study but I very strongly believe that if we corrected for this
then the curve would look like that it would be almost flat and I don't know
maybe around five six seven eight hundred total cholesterol that it might
go up for some other reason but I think the majority of people with cholesterol in the three to four hundred range if we
correct for this which we can control through lifestyle then we will not have
an increased risk from cholesterol and please hear me when I say that I'm not
suggesting that 350 is any better than 250 what I am saying the number of total
cholesterol by itself is not what we want to look at now let's just go back to the actual causes that we talked
about and see why do they cause heart disease or why is part of the reason so when we have insulin resistance chronic
inflammation oxidative stress and poor metabolic Health what happens in the
body is that the LDL changes it's not that LDL is bad but we can
change LDL into something bad if we have these factors present because LDL is
normally relatively large I call it large and fluffy that's how your liver
produces it large and fluffy it actually comes from even larger
lipoproteins that that shrink down and then if they're healthy they kind of fall into this range but if we have
these factors these destructive factors present in the body then they hurt they
damage these LDL particles and they shrink so now this inflammation causes
damage the inside of your blood vessels plus it makes these
small ldls unrecognizable you can't recycle them properly and they're small
enough to go to the wrong places and create damage
so what I have found in clinical cases we have done hundreds
of these we do blood work on everyone we test the size of these in everyone so we
can follow up and see what the changes are and when we put people on a
lifestyle change where they eat no sugar and no seed oils no processed damaged
vegetable oils we give them a high quality diet meaning good quality meat
fish chicken good quality vegetables low carb vegetables non-starchy vegetables
and we combine that with intermittent fasting meaning we restrict their
feeding windows so they don't eat every hour of every day we feed the meat vegetables and yes Eggs
unless they are sensitive to eggs then we take them out and we also give them
high quality fats we give them extra virgin olive oil lots of butter lard
Tallow coconut oil Etc stable saturated fats plus of course
extra virgin olive oil which is mostly mono unsaturated
then what we find is we measure these sizes before and after and we find that
it goes the other way that by making these lifestyle changes your reduced
inflammation and the oxidative stress and the insulin resistance and now the body can start to slowly cycle out clean
out these small damaged ldls and then the body as it makes new and healthy
ones then the proportion of these small ones get fewer and fewer lesser and less
proportion but then of course the question is does that always work does this work a hundred percent of the time
and the answer is no nothing works a hundred percent of the time and I put
only here kind of as a joke because 90 to 95 percent is Sensational and yet
that is kind of where we're where we're getting maybe not in the first couple of months maybe not the first time around
for a lot of people probably two-thirds to 75 percent they get
dramatic changes like the first time through but for others we might have to
play around with it and figure out what they're sensitive to is there an autoimmunity thing is there other things
causing inflammation but we need to put this in perspective the only 90 to 95 percent because
there's something called Placebo and in a double blind can placebo-controlled
study they always have different groups and they give one group a placebo
meaning something that looks like it's going to do something but there's nothing in there there's no active
component in there and yet the placebo group always gets somewhere around 35
percent Improvement 35 percent of people notice Improvement even though they
didn't get anything so 35 percent is kind of the Baseline that's what you get
no matter what because when people believe that they're going to get better they get better
and a success medication only has to be better than placebo
so if it's a very large study all they have to show is that it's statistically significant that it's better than
Placebo so one or two or three or five percent may be better than Placebo and
that's a sensational success for a medication and if a medication can show
maybe a 40 Improvement then they are super happy with that that's a
sensational result but we of course are not happy with 40 so we try to always
get as close to a hundred as we can so now we want to start looking at other
sources of inflammation and this is where it gets tricky because now it gets a little more complicated and a little
bit more individual variation but there are things like allergies there is other
toxicity there is autoimmunity and of course gut issues if you have
inflammation in the gut if you have poor absorption if you have leaky gut now we
have lots of variables that might be hard to control at least in the short term that starts affecting these numbers
but if we're patient enough and we're willing to continue to make changes and
monitor blood work and and check on these then very often we can get to 98
99 and that doesn't mean necessarily complete remission or that there are no
symptoms left but that we are much better off than we were and then there is that last one to two percent and this
is very unfortunate but some people are just given some bad cards that there are
various genetic factors that if you do all the things we talked about
you're still going to be way better off but that may still not be enough so the
shocking truth about eggs and heart disease is that you have been lied to we
have all been lied to for much too long for decades they have been focusing all
their attention on the wrong guy they've been demonizing the hero because it's
not about total cholesterol it's not about dietary cholesterol it's not about
saturated fats or how many eggs you eat it is about metabolic health and all the
factors that go into that like insulin toxicity gut health and the list goes on
and on and that's why we need to understand more about how the body works and how the world has changed because
the world has changed more in the last 50 years than it has in the previous fifty thousand and we need to understand
how does that affect us so that we don't just walk like lamps to the slaughter
so the solution is learn what real food is and eat that and this includes eggs
as many as you like make sure to get good quality eggs get the pastured eggs
and eat both the yolk and the egg white unless you have been found to be
sensitive or allergic to eggs because then they're going to create inflammation and then they're bad for
you so everyone is different learn what works for you but understand it from the
mechanisms and the physiology if you enjoyed this video you're going to love that one and if you truly want to master
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